Anaesthesia, Pain, Intensive Care and Emergency Medicine - A.P.I.C.E.: Proceedings of the 12th Postgraduate Course in Critical Care Medicine Trieste, Italy - November 19–21, 1997Antonio Gullo Springer Science & Business Media, 2012. gada 6. dec. - 712 lappuses The management of critically ill patients has,in the past few years, been a discipline at the forefront of development which continues to make progress with the support of basic and clinical research in the broadest sense. The application of biotechnology in this particular area has revealed itself to be essential in an attempt to provide the best interpretation of the bio-humoral and functional alterations present in a long series of often complex clinical conditions. The care of the critically ill is also at the forefront in the application of increasingly sophisticated clinical monitoring systems that also contribute to standardizing certain procedures, establish guidelines, and evaluate the efficacy of therapeutic interventions and their costs. |
No grāmatas satura
1.–5. rezultāts no 85.
. lappuse
... increase in sTNFR concentrations by 3 h, with peak plasma concentrations 10 times that of TNFα [10]. In experimental ... increased and correlate with the severity of illness and mortality [12, 13]. Interleukin1. IL1 shares many ...
... increase in sTNFR concentrations by 3 h, with peak plasma concentrations 10 times that of TNFα [10]. In experimental ... increased and correlate with the severity of illness and mortality [12, 13]. Interleukin1. IL1 shares many ...
. lappuse
... increased after endotoxin administration, and peaked at 3–4 h [21]. Interleukin6. IL6 is a cytokine having multiple ... increase in mucosal IL6. In a murine sepsis model, a significant rise in IL6 levels occurred 4 hours after CLP [31] ...
... increased after endotoxin administration, and peaked at 3–4 h [21]. Interleukin6. IL6 is a cytokine having multiple ... increase in mucosal IL6. In a murine sepsis model, a significant rise in IL6 levels occurred 4 hours after CLP [31] ...
. lappuse
... increases in pulmonary vascular resistance [28]. Several studies have shown that the vasoconstricting effects of LNMMA ... increase in pulmonary artery pressure induced by NOS inhibition may represent a major limitation to their clinical ...
... increases in pulmonary vascular resistance [28]. Several studies have shown that the vasoconstricting effects of LNMMA ... increase in pulmonary artery pressure induced by NOS inhibition may represent a major limitation to their clinical ...
. lappuse
... increases microvascular permeability [41–43] and potentiates the effects of several agonists that increase permeability [44, 45]. Rumbaut et al. [45] recently showed that NOS inhibition decreases capillary hydraulic conductivity — an ...
... increases microvascular permeability [41–43] and potentiates the effects of several agonists that increase permeability [44, 45]. Rumbaut et al. [45] recently showed that NOS inhibition decreases capillary hydraulic conductivity — an ...
. lappuse
... increased mortality rate was associated with an increase in tissue damage in the lung, liver, and kidney [49]. The timing of interventions may be of great importance. Although some studies [5, 52] reported that the rapid (5 to 30 mins) ...
... increased mortality rate was associated with an increase in tissue damage in the lung, liver, and kidney [49]. The timing of interventions may be of great importance. Although some studies [5, 52] reported that the rapid (5 to 30 mins) ...
Saturs
Pathophysiology of Flow Limitation | |
Flow limitation and bronchodilators in COPD and asthma | |
References | |
Citi izdevumi - Skatīt visu
Anaesthesia, Pain, Intensive Care and Emergency Medicine - A.P.I.C.E ... Antonio Gullo Priekšskatījums nav pieejams - 1997 |
Anaesthesia, Pain, Intensive Care and Emergency Medicine - A. P. I. C. E. Antonio Gullo Priekšskatījums nav pieejams - 2014 |
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