Anaesthesia, Pain, Intensive Care and Emergency Medicine - A.P.I.C.E.: Proceedings of the 12th Postgraduate Course in Critical Care Medicine Trieste, Italy - November 1921, 1997Antonio Gullo Springer Science & Business Media, 2012. gada 6. dec. - 712 lappuses The management of critically ill patients has,in the past few years, been a discipline at the forefront of development which continues to make progress with the support of basic and clinical research in the broadest sense. The application of biotechnology in this particular area has revealed itself to be essential in an attempt to provide the best interpretation of the bio-humoral and functional alterations present in a long series of often complex clinical conditions. The care of the critically ill is also at the forefront in the application of increasingly sophisticated clinical monitoring systems that also contribute to standardizing certain procedures, establish guidelines, and evaluate the efficacy of therapeutic interventions and their costs. |
No grāmatas satura
1.5. rezultāts no 38.
. lappuse
... hepatic nitrogen metabolism Acidbase regulation of urea synthesis Pathophysiological aspects References AcidBase Disturbances during Fulminant Hepatic Failure The liver in acidbase homeostasis Acid base disorders during Fulminant Hepatic ...
... hepatic nitrogen metabolism Acidbase regulation of urea synthesis Pathophysiological aspects References AcidBase Disturbances during Fulminant Hepatic Failure The liver in acidbase homeostasis Acid base disorders during Fulminant Hepatic ...
. lappuse
... hepatic arterial resistance increased after LNAME under control and endotoxic shock conditions in pigs thus reducing total hepatic blood flow and diminishing venous return across the liver. Mulder et al. [11] partially (0.1 mg) or ...
... hepatic arterial resistance increased after LNAME under control and endotoxic shock conditions in pigs thus reducing total hepatic blood flow and diminishing venous return across the liver. Mulder et al. [11] partially (0.1 mg) or ...
. lappuse
... (hepatic in particular). Unlike the cNOS isoforms that are tightly regulated by calcium levels, the iNOS enzyme is limited only by the quantity of active iNOS protein, the availability of cofactors, and the availability of.
... (hepatic in particular). Unlike the cNOS isoforms that are tightly regulated by calcium levels, the iNOS enzyme is limited only by the quantity of active iNOS protein, the availability of cofactors, and the availability of.
. lappuse
... hepatic, portal and mesenteric blood flow in a dog model of endotoxic shock [19]. In endotoxic rabbits, Pastor et al. [36] reported that SIN1 maintained aortic artery blood flow and portal vein blood flow, and increased hepatic artery ...
... hepatic, portal and mesenteric blood flow in a dog model of endotoxic shock [19]. In endotoxic rabbits, Pastor et al. [36] reported that SIN1 maintained aortic artery blood flow and portal vein blood flow, and increased hepatic artery ...
. lappuse
... hepatic perfusion while preventing lactic acidosis. The protective effects of NO donors on the liver in endotoxemia is associated with an upregulation of hepatic protein synthesis by NO donors [86]. BoughtonSmith et al. [87] reported ...
... hepatic perfusion while preventing lactic acidosis. The protective effects of NO donors on the liver in endotoxemia is associated with an upregulation of hepatic protein synthesis by NO donors [86]. BoughtonSmith et al. [87] reported ...
Saturs
Pathophysiology of Flow Limitation | |
Flow limitation and bronchodilators in COPD and asthma | |
References | |
Citi izdevumi - Skatīt visu
Anaesthesia, Pain, Intensive Care and Emergency Medicine - A.P.I.C.E ... Antonio Gullo Priekšskatījums nav pieejams - 1997 |
Anaesthesia, Pain, Intensive Care and Emergency Medicine - A. P. I. C. E. Antonio Gullo Priekšskatījums nav pieejams - 2014 |
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abdominal acidbase acidosis acute administration airway alveolar analgesia Anesth Analg anesthesia Anesthesiology anesthetic aortic ARDS arterial pressure barotrauma blood flow blood pressure brain cardiac cardiovascular cells cerebral clinical cm H2O coronary critically ill cytokines decrease diabetic disease dobutamine dose drugs dysfunction edema effects endotoxemia endotoxic endotoxic shock endotoxin epidural evaluation failure fluid function heart hemodynamic hemofiltration hemorrhage hepatic hypertension hypotension hypothermia increase induced infection inflammatory infusion inhibition inhibitors intensive intravenous intubation ischemic lactate levels liver lung injury mechanical ventilation mediators metabolic monitoring mortality muscle myocardial ischemia nitric oxide nitric oxide synthase opioids outcome oxygen pain patients PEEP perfusion perioperative Physiol physiological plasma postoperative preoperative propacetamol propofol pulmonary receptors reduced remifentanil renal Respir Crit respiratory response risk role sepsis septic shock surfactant surgery surgical syndrome synthesis temperature therapy tidal volume tissue tramadol transplantation trauma treatment urea vascular vasodilation venous ventilatory ventricular