Anaesthesia, Pain, Intensive Care and Emergency Medicine - A.P.I.C.E.: Proceedings of the 12th Postgraduate Course in Critical Care Medicine Trieste, Italy - November 19–21, 1997Antonio Gullo Springer Science & Business Media, 2012. gada 6. dec. - 712 lappuses The management of critically ill patients has,in the past few years, been a discipline at the forefront of development which continues to make progress with the support of basic and clinical research in the broadest sense. The application of biotechnology in this particular area has revealed itself to be essential in an attempt to provide the best interpretation of the bio-humoral and functional alterations present in a long series of often complex clinical conditions. The care of the critically ill is also at the forefront in the application of increasingly sophisticated clinical monitoring systems that also contribute to standardizing certain procedures, establish guidelines, and evaluate the efficacy of therapeutic interventions and their costs. |
No grāmatas satura
1.–5. rezultāts no 81.
. lappuse
... administration of exogenous TNF reproduces many of the features of clinical sepsis [3,5,6]. TNF administration to humans initiates many of the characteristic components of the inflammatory stress response [5–8]. In addition to acute ...
... administration of exogenous TNF reproduces many of the features of clinical sepsis [3,5,6]. TNF administration to humans initiates many of the characteristic components of the inflammatory stress response [5–8]. In addition to acute ...
. lappuse
... administration, but plasma sTNFR peaked at 3 h. In patients with sepsis, plasma sTNFR concentration are increased and correlate with the severity of illness and mortality [12, 13]. Interleukin1. IL1 shares many proinflammatory ...
... administration, but plasma sTNFR peaked at 3 h. In patients with sepsis, plasma sTNFR concentration are increased and correlate with the severity of illness and mortality [12, 13]. Interleukin1. IL1 shares many proinflammatory ...
. lappuse
... administration, and peaked at 3–4 h [21]. Interleukin6. IL6 is a cytokine having multiple biological activities on a wide variety of cells [22]. IL6 is found in serum and other bodily fluids as a result of severe infection, inflammation ...
... administration, and peaked at 3–4 h [21]. Interleukin6. IL6 is a cytokine having multiple biological activities on a wide variety of cells [22]. IL6 is found in serum and other bodily fluids as a result of severe infection, inflammation ...
. lappuse
... administration, whereas TNF concentrations peaked at 1 to 2 h [42]. Like IL6, IL8 production in sepsis appears to be activated by TNF. Redl et al. [44] showed that pretreatment of baboons with antiTNF antibodies can decrease plasma IL8 ...
... administration, whereas TNF concentrations peaked at 1 to 2 h [42]. Like IL6, IL8 production in sepsis appears to be activated by TNF. Redl et al. [44] showed that pretreatment of baboons with antiTNF antibodies can decrease plasma IL8 ...
. lappuse
... administration of tumor necrosis factor to normal subjects. N Engl J Med 322:1622–1627 8. Warren RS, Starnes Jr HF, Gabrilove JL et al (1987) The acute metabolic effects of tumor necrosis factor administration in humans. Arch Surg 122 ...
... administration of tumor necrosis factor to normal subjects. N Engl J Med 322:1622–1627 8. Warren RS, Starnes Jr HF, Gabrilove JL et al (1987) The acute metabolic effects of tumor necrosis factor administration in humans. Arch Surg 122 ...
Saturs
Pathophysiology of Flow Limitation | |
Flow limitation and bronchodilators in COPD and asthma | |
References | |
Citi izdevumi - Skatīt visu
Anaesthesia, Pain, Intensive Care and Emergency Medicine - A.P.I.C.E ... Antonio Gullo Priekšskatījums nav pieejams - 1997 |
Anaesthesia, Pain, Intensive Care and Emergency Medicine - A. P. I. C. E. Antonio Gullo Priekšskatījums nav pieejams - 2014 |
Bieži izmantoti vārdi un frāzes
abdominal acidbase acidosis acute administration airway alveolar analgesia Anesth Analg anesthesia Anesthesiology anesthetic aortic ARDS arterial pressure barotrauma blood flow blood pressure brain cardiac cardiovascular cells cerebral clinical cm H2O coronary critically ill cytokines decrease diabetic disease dobutamine dose drugs dysfunction edema effects endotoxemia endotoxic endotoxic shock endotoxin epidural evaluation failure fluid function heart hemodynamic hemofiltration hemorrhage hepatic hypertension hypotension hypothermia increase induced infection inflammatory infusion inhibition inhibitors intensive intravenous intubation ischemic lactate levels liver lung injury mechanical ventilation mediators metabolic monitoring mortality muscle myocardial ischemia nitric oxide nitric oxide synthase opioids outcome oxygen pain patients PEEP perfusion perioperative Physiol physiological plasma postoperative preoperative propacetamol propofol pulmonary receptors reduced remifentanil renal Respir Crit respiratory response risk role sepsis septic shock surfactant surgery surgical syndrome synthesis temperature therapy tidal volume tissue tramadol transplantation trauma treatment urea vascular vasodilation venous ventilatory ventricular